Introduction
- A raised blood concentration of lipid in the fasted (>12 hours) patient exceeding the upper range of normal.
- Cause : primary idiopathic, or secondary to another disease process.
- Signs : may be asymptomatic; seizures, abdominal pain, distress, ocular involvement; secondary hyperlipidemia - signs of primary causative disease process.
- Diagnosis : biochemistry - serum triglyceride, lipid and cholesterol concentrations; lipoprotein electrophoresis.
- Treatment : reduce dietary fat and/or treatment of primary disease process.
- Prognosis : dependent upon response to treatment of primary disease process.
Diagnosis
Clinical signs
- Lipemia retinalis .
- Lipemic aqueous.
- Neuropathies.
Diagnosis
Differential diagnosis
- Post-prandial hyperlipidemia.
Primary hyperlipidemia
- Idiopathic hyperchylomicronemia (Himalayan).
Ocular hyperlipidemia
- Same as presenting problem -Cloudy eye.
- Same as presenting problem - Blindness.
Sequelae
Prognosis
- Acute fatal pancreatitis
may occur secondary to hyperlipidemia. - Secondary hyperlipidemia - prognosis depends on response to treatment of primary disease.
Expected response to treatment
- Peripheral neuropathies usually resolve 2-3 months after institution of low fat diet + successful treatment of other primary disease.
Reasons for treatment failure
- Incorrect diagnosis.
- Owner non-compliance in treating animal.
Sources
Publications
Refereed papers
- Bauer J E (1997)New concepts of polyunsaturated fatty acids in dogs and cats.Vet Clin Nutrition4(1), 29-33.
- Whitney M S (1992)Evaluation of hyperlipidemias in dogs and cats.Semin Vet Med Surg Small Anim7(4), 292-300.
Other sources of information
- Bauer J E (2000)Hyperlipidemias.In:Textbook of Veterinary Internal Medicine.5th edn. Ettinger S J & Feldman E C (eds). W B Saunders, Philadelphia. pp 283-292.



