Introduction
- Congenital or acquired vascular anomaly in which portal blood bypasses the liver → systemic circulation via portal vein.
- Signs :
- Gastrointestinal - vomiting, diarrhea, anorexia, ptyalism.
- Central nervous system (CNS) - depression/hyperactivity, head pressing, pacing/circling, stupor, seizures, coma.
- Other - weight loss, polyuria/polydipsia, ascites, failure to metabolize sedatives and other drugs, urolithiasis.
- Diagnosis : history, age, bile salt analysis, radiography, ultrasonography.
- Treatment : surgical ligation of shunt; medical management in acute cases prior to surgery, in long-term cases where surgery impossible/failed/declined by owners.
- Prognosis : guarded.
Diagnosis
Clinical signs
- Ataxia.
- Ptyalism.
- Hyperactivity.
- Pyrexia.
- Ascites.
- Pleural fluid.
- Coma, stupor.
Diagnosis
Differential diagnosis
Sequelae
Prognosis
- Good: if surgery successful.
- Reasonable: if managed medically - can survive long periods.
Reasons for treatment failure
- Failure to locate or ligate shunting vessel(s).
- Acute portal hypertension following surgery.
- Failure to respond to medical management.
Sources
Publications
Refereed papers
- Tillson DM & Winkler JT (2002)Diagnosis and treatment of portosystemic shunts in the cat.Vet Clin North Am Small Anim Pract.32(4), 881-899.
- Langdon Pet al(2002)Acquired portosystemic shunting in two cats.J Am Anim Hosp Assoc.38(1), 21-27.
- Heldmann Eet al(1999)Use of propofol to manage seizure activity after surgical treatment of portosystemic shunts.JSAP40, (12) 590-4.
- Birchard S J & Sherding R G (1992)Feline portosystemic shunts.Comp Cont Educ Pract Vet14, 1295.
Other sources of information
- Levy J K, Bunch S E & Komtebede J (1995)Feline portosystemic shunts.In:Kirk's Current Veterinary Therapy XII.W B Saunders. pp 743-749.




