Introduction
- Cause : virus disease of the central nervous system (CNS).
- Signs : 'furious' and 'dumb' forms.
- All mammals susceptible (to a variable extent).
- Reservoir hosts vary with country.
- Treatment : none.
- Diagnosis : signs, confirmation on examination of brain at histopathology.
- Prognosis : grave/hopeless.
Diagnosis
Clinical signs
Prodromal phase
- Slight fever.
- Pupillary dilation.
- After salivary secretion of virus begins, progresses to dumb or to furious rabies.
- Lick, chew or mutilate original bite site.
- May follow prodromal stage (75%), or follow 'furious' rabies.
- Change in vocalization: howling, bellowing.
- Paralysis of lower jaw and eyelids (squinting).
- Drooling of saliva.
- Weakness of limbs, tail.
- Difficulty swallowing.
- Hydrophobia is NOT a feature (only seen in man).
- Coma → death.
'Furious' rabies
- 25% of cases.
- Aggression.
- Drooling of saliva.
- Attack without provocation.
- Disordered wandering.
- Intermittent hyperexcitability.
- Depraved appetite/pica → gastritis.
- Altered vocalization (howling).
- Progresses to either 'dumb' rabies (see above), or inco-ordination, convulsions, coma, death.
- Sensitive to noise → attack.
- Some dogs have mild signs, recover, and remain as persistent excretors.
Sequelae
Prognosis
- Grave/hopeless.
- Some dogs with mild signs may recover, but remain persistent carriers.
Expected response to treatment
- Gradual progression to death within 5-15 days of onset of signs.
Reasons for treatment failure
- All affected dogs should be euthanased.
Sources
Publications
Refereed papers
- Recent references from PubMed.
Other sources of information
- Beynon P H & Edney A T B (1995) Rabies in a Changing World. In: Proceedings of a Joint Symposium held at the Royal Society of Medicine, London. 3rd May. Cheltenham: British Small Animal Veterinary Association.




